Jerry Bergman is a famous creationist author who has extensively published over many decades and who has taught at several universities. He describes the research that went into this book:

“This project has been for me a lifelong study for which this book is a summary. It is the culmination of four decades of research on the issue of evolution, 41 years of teaching life science at the college level, and over 1,700 publications in 2,400 college libraries in 65 nations and 13 languages” (p. xvii).

In terms of specifics, this work focuses on taxonomy, so-called convergent evolution, irreducible complexity, pseudogenes, and antibiotic resistance. Bergman goes into considerable detail on all of these.

Bacterial resistance to antibiotics: not evolutionary novelties

Evolutionists often cite antibiotic resistance in bacteria as evidence for evolution. It is not. It is a product of the ‘tweaking’ of pre-existing features in the bacteria, which raises the question of how these features arose in the first place

Bergman has examined and deconstructed a variety of ‘pillars’ of evolution. Each one of them contains major flaws. Neither taxonomy nor genetics, for example, compel belief in evolution.

Irreducible Complexity

Evolutionists have caricatured, but not overcome, the fatal problem of irreducible complexity. The best explanation for living things, whether somebody likes it or not, remains an intelligent designer.

Evolutionists have argued that biological systems only appear to be irreducibly complex because these systems once possessed numerous redundancies that enabled the components to function independently from each other. These redundancies have since been removed by evolutionary processes, leaving the remaining components in a state of lockstep dependence upon each other—hence the apparent irreducible complexity.

To begin with, the explanation is ad hoc. There is no evidence for any such one-time grand redundancies, and, if they are going to pooh-pooh the Intelligent Design explanation, the burden of proof is on the evolutionists to show that they once existed. Note also that spot redundancies should not be confused with the hypothesized grand redundancies that presumably governed the whole. In the Krebs cycle, for example, a few of the compounds can be synthesized by alternative pathways. The fact that parts of the Krebs cycle are redundant is very different from saying that the Krebs cycle as a whole is, or once was, redundant. The fact that there are ‘shortcuts’ within the Krebs cycle is very different from suggesting that the entire Krebs cycle can be bypassed by a shortcut. Bergman quips: “Reducing the cycle by one step does not negate the fact that it still requires the remaining parts of the cycle. It would not be irreducibly complex only if a single quark were responsible for the biochemical results that the Krebs cycle achieves” (p. 123).

An analogy with the automobile may help. The car thief can do a ‘shortcut’ around the key-starting system by short-circuiting the wires that lead to the starter motor and driving away the car. This means that, from a mechanical point of view, the keystarting system is redundant. However, this individual redundancy certainly does not mean that the automobile as a whole is a redundant system, much less that the automobile could spontaneously arise, step-by-step, without intelligent design.